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MTOR-independent autophagy induced by interrupted endoplasmic reticulum-mitochondrial Ca<sup>2+</sup> communication: a dead end in cancer cells.

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第一作者： Ulises,Ahumada-Castro

第一单位： a Center for Integrative Biology, Faculty of Sciences , Universidad Mayor , Santiago , Chile.;b Geroscience Center for Brain Health and Metabolism , Santiago , Chile.

作者： Ulises,Ahumada-Castro ^[1] ; Eduardo,Silva-Pavez ^[1] ; Alenka,Lovy ^[2] ; Evelyn,Pardo ^[3] ; Jordi,Molgό ^[4] ; César,Cárdenas ^[5]

作者单位： a Center for Integrative Biology, Faculty of Sciences , Universidad Mayor , Santiago , Chile.;b Geroscience Center for Brain Health and Metabolism , Santiago , Chile. ^[1] c Department of Neuroscience , Center for Neuroscience Research, Tufts School of Medicine , Boston , MA , USA. ^[2] b Geroscience Center for Brain Health and Metabolism , Santiago , Chile. ^[3] d CEA, Institut des Sciences du Vivant Frédéric Joliot, Université Paris-Saclay , Service d'Ingénierie Moléculaire des Protéines , Gif-sur-Yvette , France.;e Anatomy and Developmental Biology Program , Institute of Biomedical Science, University of Chile , Santiago , Chile. ^[4] b Geroscience Center for Brain Health and Metabolism , Santiago , Chile.;e Anatomy and Developmental Biology Program , Institute of Biomedical Science, University of Chile , Santiago , Chile.;f The Buck Institute for Research on Aging , Novato , CA , USA.;g Department of Chemistry and Biochemistry , University of California , Santa Barbara , CA , USA. ^[5]

关键词 4 5-triphosphate receptors AMPK ITPRs/inositol 1 MCU mitochondria associated membranes