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Paired Immunoglobulin-like Type 2 Receptor Alpha G78R variant alters ligand binding and confers protection to Alzheimer's disease.

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第一作者： Nisha,Rathore

第一单位： Department of OMNI Human Genetics, Genentech Inc., South San Francisco, California, United States of America.

作者： Nisha,Rathore ^[1] ; Sree Ranjani,Ramani ^[2] ; Homer,Pantua ^[3] ; Jian,Payandeh ^[4] ; Tushar,Bhangale ^[5] ; Arthur,Wuster ^[5] ; Manav,Kapoor ^[6] ; Yonglian,Sun ^[7] ; Sharookh B,Kapadia ^[3] ; Lino,Gonzalez ^[8] ; Ali A,Zarrin ^[7] ; Alison,Goate ^[6] ; David V,Hansen ^[9] ; Timothy W,Behrens ^[1] ; Robert R,Graham ^[1]

作者单位： Department of OMNI Human Genetics, Genentech Inc., South San Francisco, California, United States of America. ^[1] Department of Microchemistry, Proteomics & Lipidomics, Genentech Inc., South San Francisco, California, United States of America. ^[2] Department of Immunology and Infectious Diseases, Genentech Inc., South San Francisco, California, United States of America. ^[3] Department of Structural Biology, Genentech Inc., South San Francisco, California, United States of America. ^[4] Department of OMNI Human Genetics, Genentech Inc., South San Francisco, California, United States of America.;Department of Bioinformatics and Computational Biology, Genentech Inc., South San Francisco, California, United States of America. ^[5] Department of Neuroscience, Ronald M. Loeb Center for Alzheimer's disease, Icahn School of Medicine at Mount Sinai, New York, United States of America. ^[6] Department of Immunology, Genentech Inc., South San Francisco, California, United States of America. ^[7] Department of Proteomics & Biological Resources, Genentech Inc., South San Francisco, California, United States of America. ^[8] Department of Neuroscience, Genentech Inc., South San Francisco, California, United States of America. ^[9]