Matrix metalloproteinase-12 as an endogenous resolution promoting factor following myocardial infarction.

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第一作者： Alan J,Mouton

第一单位： Mississippi Center for Heart Research, Department of Physiology and Biophysics, University of Mississippi Medical Center, 2500 N State St, Jackson, MS, 39216, United States.

作者： Alan J,Mouton ^[1] ; Osvaldo J,Rivera Gonzalez ^[1] ; Amanda R,Kaminski ^[1] ; Edwin T,Moore ^[1] ; Merry L,Lindsey ^[2]

作者单位： Mississippi Center for Heart Research, Department of Physiology and Biophysics, University of Mississippi Medical Center, 2500 N State St, Jackson, MS, 39216, United States. ^[1] Mississippi Center for Heart Research, Department of Physiology and Biophysics, University of Mississippi Medical Center, 2500 N State St, Jackson, MS, 39216, United States; Research Service, G.V. (Sonny) Montgomery Veterans Affairs Medical Center, 1500 E Woodrow Wilson Ave, Jackson, MS, 39216, United States. Electronic address: mllindsey@umc.edu. ^[2]

关键词 Extracellular matrix Fibroblast Inflammation Inflammation resolution Macrophage Neutrophil

主题词动物(Animals);人类(Humans);基质金属蛋白酶12(Matrix Metalloproteinase 12);心肌梗死(Myocardial Infarction)

DOI 10.1016/j.phrs.2018.10.026

PMID 30394317

发布时间 2019-11-01

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