Crosstalk between Raf-MEK-ERK and PI3K-Akt-GSK3β signaling networks promotes chemoresistance, invasion/migration and stemness via expression of CD44 variants (v4 and v6) in oral cancer.
作者:
主题词
成年人(Adult);老年人(Aged);抗肿瘤药(Antineoplastic Agents);细胞系, 肿瘤(Cell Line, Tumor);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);顺铂(Cisplatin);抗药性, 肿瘤(Drug Resistance, Neoplasm);女(雌)性(Female);基因敲低技术(Gene Knockdown Techniques);人类(Humans);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);中年人(Middle Aged);口腔肿瘤(Mouth Neoplasms);肿瘤侵润(Neoplasm Invasiveness);肿瘤干细胞(Neoplastic Stem Cells);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);蛋白质亚型(Protein Isoforms);蛋白激酶抑制剂(Protein Kinase Inhibitors);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);RNA, 小分子干扰(RNA, Small Interfering)
DOI
10.1016/j.oraloncology.2018.09.028
PMID
30409306
发布时间
2019-12-17
- 浏览11
Oral oncology
234-243页
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