Persistent IKKα phosphorylation induced apoptosis in UVB and Poly I:C co-treated HaCaT cells plausibly through pro-apoptotic p73 and abrogation of IκBα.

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第一作者： Wuxiyar,Otkur

第一单位： China-Japan Research Institute of Medical and Pharmaceutical Sciences, Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, 110016, China.

作者： Wuxiyar,Otkur ^[1] ; Fang,Wang ^[1] ; Weiwei,Liu ^[1] ; Toshihiko,Hayashi ^[2] ; Shin-Ichi,Tashiro ^[3] ; Satoshi,Onodera ^[4] ; Takashi,Ikejima ^[5]

作者单位： China-Japan Research Institute of Medical and Pharmaceutical Sciences, Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, 110016, China. ^[1] China-Japan Research Institute of Medical and Pharmaceutical Sciences, Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, 110016, China; Department of Chemistry and Life Science, School of Advanced Engineering, Kogakuin University, 2665-1, Nakanomachi, Hachioji, Tokyo, 192-0015, Japan. ^[2] Department of Medical Education & Primary Care, Kyoto Prefectural University of Medicine, Kajiicho 465, Kamikyo-ku, Kyoto City, Kyoto, 602-8566, Japan. ^[3] Department of Clinical and Pharmaceutical Sciences, Showa Pharmaceutical University, Tokyo, 194-8543, Japan. ^[4] China-Japan Research Institute of Medical and Pharmaceutical Sciences, Wuya College of Innovation, Shenyang Pharmaceutical University, Shenyang, 110016, China. Electronic address: ikejimat@vip.sina.com. ^[5]

关键词 Apoptosis IKKα IκBα Toll-like receptor 3 UVB p73