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JAK2V617F but not CALR mutations confer increased molecular responses to interferon-α via JAK1/STAT1 activation.

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第一作者: Julia,Czech
第一单位: Department of Hematology, Oncology, Hemostaseology, and Stem Cell Transplantation, Faculty of Medicine, RWTH Aachen University, Aachen, Germany.
作者单位: Department of Hematology, Oncology, Hemostaseology, and Stem Cell Transplantation, Faculty of Medicine, RWTH Aachen University, Aachen, Germany. [1] Department of Hematology, Zealand University Hospital, Copenhagen University Hospital, Roskilde, Denmark. [2] Department of Internal Medicine, Hematology and Oncology, Masaryk University and University Hospital Brno, Brno, Czech Republic. [3] Institute for Computational Genomics, Joint Research Center for Computational Biomedicine, RWTH Aachen University, Aachen, Germany. [4] Interdisciplinary Center for Clinical Research Aachen Faculty of Medicine, RWTH Aachen University, Aachen, Germany. [5] Department of Hematology, Oncology, Hemostaseology, and Stem Cell Transplantation, Faculty of Medicine, RWTH Aachen University, Aachen, Germany.;Institute of Pathology, Faculty of Medicine, RWTH Aachen University, Aachen, Germany. [6] Institute of Biochemistry and Molecular Biology, Faculty of Medicine, RWTH Aachen University, Aachen, Germany. [7] Department of Hematology, Odense University Hospital, Odense, Denmark. [8] Department of Hematology, Oncology, Hemostaseology, and Stem Cell Transplantation, Faculty of Medicine, RWTH Aachen University, Aachen, Germany. skoschmieder@ukaachen.de. [9]
DOI 10.1038/s41375-018-0295-6
PMID 30470838
发布时间 2022-04-20
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