Mycoplasma promotes malignant transformation in vivo, and its DnaK, a bacterial chaperone protein, has broad oncogenic properties.

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作者： Davide,Zella ^[1] ; Sabrina,Curreli ^[2] ; Francesca,Benedetti ^[3] ; Selvi,Krishnan ^[4] ; Fiorenza,Cocchi ^[3] ; Olga S,Latinovic ^[2] ; Frank,Denaro ^[3] ; Fabio,Romerio ^[3] ; Muhammad,Djavani ^[4] ; Man E,Charurat ^[3] ; Joseph L,Bryant ^[5] ; Hervé,Tettelin ^[6] ; Robert C,Gallo ^[3]

作者单位： Institute of Human Virology, School of Medicine, University of Maryland, Baltimore, MD 21201; dzella@ihv.umaryland.edu rgallo@ihv.umaryland.edu. ^[1] Department of Biochemistry and Molecular Biology, School of Medicine, University of Maryland, Baltimore, MD 21201. ^[2] Institute of Human Virology, School of Medicine, University of Maryland, Baltimore, MD 21201. ^[3] Department of Medicine, School of Medicine, University of Maryland, Baltimore, MD 21201. ^[4] Department of Microbiology and Immunology, School of Medicine, University of Maryland, Baltimore, MD 21201. ^[5] Department of Biology, Morgan State University, Baltimore, MD 21251. ^[6] Department of Pathology, School of Medicine, University of Maryland, Baltimore, MD 21201. ^[7] Institute for Genome Sciences, School of Medicine, University of Maryland, Baltimore, MD 21201. ^[8]

关键词 DNA repair DnaK cancer mycoplasma p53