Transactivation of RAGE mediates angiotensin-induced inflammation and atherogenesis.

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作者： Raelene J,Pickering ^[1] ; Christos,Tikellis ^[2] ; Carlos J,Rosado ^[1] ; Despina,Tsorotes ^[2] ; Alexandra,Dimitropoulos ^[1] ; Monique,Smith ^[2] ; Olivier,Huet ^[2] ; Ruth M,Seeber ^[1] ; Rekhati,Abhayawardana ^[1] ; Elizabeth Km,Johnstone ^[2] ; Jonathan,Golledge ^[3] ; Yutang,Wang ^[4] ; Karin A,Jandeleit-Dahm ^[4] ; Mark E,Cooper ^[4] ; Kevin Dg,Pfleger ^[5] ; Merlin C,Thomas ^[5]

作者单位： Department of Diabetes, Central Clinical School, Monash University, Melbourne, Australia. ^[1] Baker IDI Heart and Diabetes Institute, Melbourne, Australia. ^[2] Department of Anaesthesia and Intensive Care, Centre Hospitalier Régional Universitaire (CHRU) La Cavale Blanche, Université de Bretagne Ouest, Brest, France. ^[3] Molecular Endocrinology and Pharmacology, Harry Perkins Institute of Medical Research and Centre for Medical Research, The University of Western Australia, Nedlands, Australia. ^[4] The Vascular Biology Unit, Queensland Research Centre for Peripheral Vascular Disease, James Cook University, Townsville, Australia. ^[5] Dimerix Limited, Nedlands, Western Australia, Australia. ^[6]

关键词 Atherosclerosis Cell Biology G-protein coupled receptors NF-kappaB Vascular Biology