换一批Bcl-2 inhibitors enhance FGFR inhibitor-induced mitochondrial-dependent cell death in FGFR2-mutant endometrial cancer.
第一作者：
Leisl M,Packer
第一单位：
School of Biomedical Science, Institute of Health & Biomedical Innovation, Queensland University of Technology located within the Translational Research Institute, Brisbane, Australia.
作者：
医学主题词
动物(Animals);细胞凋亡(Apoptosis);半胱氨酸天冬氨酸蛋白酶(Caspases);细胞系, 肿瘤(Cell Line, Tumor);子宫内膜肿瘤(Endometrial Neoplasms);酶激活(Enzyme Activation);女(雌)性(Female);人类(Humans);半数抑制浓度(Inhibitory Concentration 50);小鼠(Mice);线粒体(Mitochondria);突变(Mutation);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);受体, 成纤维细胞生长因子, 2型(Receptor, Fibroblast Growth Factor, Type 2)
DOI
10.1002/1878-0261.12422
PMID
30537101
发布时间
2020-03-09
基金项目
1087165/Cancer Australia/International
1011301/NHMRC/International
1032851/NHMRC/International
443038/NHMRC/International
- 浏览11
Molecular oncology
738-756页
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