Probucol protects circulating endothelial progenitor cells from ambient PM<sub>2.5</sub> damage via inhibition of reactive oxygen species and inflammatory cytokine production <i>in vivo</i>.

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第一作者： Yong,Chen

第一单位： Department of Emergency, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong 250021, P.R. China.

作者： Yong,Chen ^[1] ; Ke,Hu ^[2] ; Haoran,Bu ^[1] ; Zhihua,Si ^[3] ; Haihui,Sun ^[4] ; Liming,Chen ^[4] ; Hang,Liu ^[4] ; Hao,Xie ^[4] ; Peng,Zhao ^[4] ; Le,Yang ^[4] ; Qinghua,Sun ^[5] ; Zhenguo,Liu ^[6] ; Lianqun,Cui ^[4] ; Yuqi,Cui ^[4]

作者单位： Department of Emergency, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong 250021, P.R. China. ^[1] Department of Emergency, Qianfoshan Hospital, Shandong University, Jinan, Shandong 250021, P.R. China. ^[2] Department of Neurology, Qianfoshan Hospital, Shandong University, Jinan, Shandong 250021, P.R. China. ^[3] Department of Cardiology, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong 250021, P.R. China. ^[4] Division of Environmental Health Sciences, The Ohio State University, Columbus, OH 43210, USA. ^[5] Center for Precision Medicine and Division of Cardiovascular Medicine, University of Missouri School of Medicine, Columbia, MO 65212, USA. ^[6]

关键词 PM2.5 endothelial progenitor cells inflammatory cytokines probucol reactive oxygen species

DOI 10.3892/etm.2018.6791

PMID 30542381

发布时间 2020-09-29

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