Inflammation and NLRP3 Inflammasome Activation Initiated in Response to Pressure Overload by Ca<sup>2+</sup>/Calmodulin-Dependent Protein Kinase II δ Signaling in Cardiomyocytes Are Essential for Adverse Cardiac Remodeling.
作者:
主题词
动物(Animals);细胞凋亡(Apoptosis);钙-钙调素依赖性蛋白激酶2型(Calcium-Calmodulin-Dependent Protein Kinase Type 2);炎症趋化因子类(Chemokines);细胞因子类(Cytokines);女(雌)性(Female);纤维化(Fibrosis);心力衰竭(Heart Failure);炎性体(Inflammasomes);炎症(Inflammation);巨噬细胞(Macrophages);男(雄)性(Male);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);肌细胞, 心脏(Myocytes, Cardiac);NF-κB(NF-kappa B);活性氧(Reactive Oxygen Species);信号传导(Signal Transduction);心室重构(Ventricular Remodeling)
DOI
10.1161/CIRCULATIONAHA.118.034621
PMID
30571348
发布时间
2020-03-09
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Circulation
2530-2544页
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