Defective glycosylation leads to defective gp130-dependent STAT3 signaling in PGM3-deficient patients.

导出原文传递

翻译打印收藏纠错

作者： Meriem,Ben-Ali ^[1] ; Leila,Ben-Khemis ^[2] ; Najla,Mekki ^[1] ; Roukaya,Yaakoubi ^[1] ; Rym,Ouni ^[1] ; Chaouki,Benabdessalem ^[1] ; Imen,Ben-Mustapha ^[1] ; Mohamed-Ridha,Barbouche ^[3]

作者单位： Laboratory of Transmission, Control and Immunobiology of Infections (LR11IPT02), Institut Pasteur de Tunis, Tunis, Tunisia; University Tunis El Manar, Tunis, Tunisia. ^[1] Laboratory of Transmission, Control and Immunobiology of Infections (LR11IPT02), Institut Pasteur de Tunis, Tunis, Tunisia; University of Carthage, Sidi Bou Said, Carthage, Tunisia. ^[2] Laboratory of Transmission, Control and Immunobiology of Infections (LR11IPT02), Institut Pasteur de Tunis, Tunis, Tunisia; University Tunis El Manar, Tunis, Tunisia. Electronic address: ridha.barbouche@pasteur.rns.tn. ^[3]

主题词细胞因子受体gp130(Cytokine Receptor gp130);糖基化(Glycosylation);人类(Humans);葡糖磷酸变位酶(Phosphoglucomutase);STAT3转录因子(STAT3 Transcription Factor);信号传导(Signal Transduction)

DOI 10.1016/j.jaci.2018.12.987

PMID 30578875

发布时间 2020-04-30

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交