MiR-103 inhibiting cardiac hypertrophy through inactivation of myocardial cell autophagy via targeting TRPV3 channel in rat hearts.

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作者： Hanping,Qi ^[1] ; Jing,Ren ^[1] ; Mingyao,E ^[1] ; Qianhui,Zhang ^[1] ; Yonggang,Cao ^[1] ; Lina,Ba ^[1] ; Chao,Song ^[1] ; Pilong,Shi ^[1] ; Bowen,Fu ^[1] ; Hongli,Sun ^[1]

作者单位： Department of Pharmacology, Harbin Medical University-Daqing, Daqing, China. ^[1]

关键词 TRPV3 autophagy cardiac hypertrophy miR-103

主题词血管紧张素Ⅱ(Angiotensin II);动物(Animals);自噬(Autophagy);心脏扩大(Cardiomegaly);细胞, 培养的(Cells, Cultured);减量调节(Down-Regulation);心脏(Heart);心力衰竭(Heart Failure);男(雄)性(Male);微RNAs(MicroRNAs);肌细胞, 心脏(Myocytes, Cardiac);大鼠(Rats);大鼠, Wistar(Rats, Wistar);信号传导(Signal Transduction);TRPV阳离子通道(TRPV Cation Channels);增量调节(Up-Regulation)

DOI 10.1111/jcmm.14095

PMID 30604587

发布时间 2023-10-05

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Journal of cellular and molecular medicine

2019年23卷3期

1926-1939页

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MiR-103 inhibiting cardiac hypertrophy through inactivation of myocardial cell autophagy via targeting TRPV3 channel in rat hearts.

Journal of cellular and molecular medicine

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Journal of cellular and molecular medicine

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MiR-103 inhibiting cardiac hypertrophy through inactivation of myocardial cell autophagy via targeting TRPV3 channel in rat hearts.

Journal of cellular and molecular medicine

相似文献

Journal of cellular and molecular medicine

相关期刊

检索历史清除