换一批Natriuretic Peptide Receptor-C Protects Against Angiotensin II-Mediated Sinoatrial Node Disease in Mice.
第一作者：
Martin,Mackasey
第一单位：
Department of Cardiac Sciences, Libin Cardiovascular Institute of Alberta, Cumming School of Medicine, University of Calgary, Calgary, Alberta, Canada, and Department of Physiology and Pharmacology, Cumming School of Medicine, University of Calgary, Calgary, Alberta, Canada.;Department of Physiology and Biophysics, Faculty of Medicine, Dalhousie University, Halifax, Nova Scotia, Canada.
作者：
关键词
AP, action potentialAng II, angiotensin IICV, conduction velocityDD, diastolic depolarizationGmax, maximum conductanceHR, heart rateICa,L, L-type calcium currentICa,T, T-type calcium currentINCX, sodium–calcium exchanger currentIV, current voltage relationshipIf, hyperpolarization-activated currentNP, natriuretic peptideNPR, natriuretic peptide receptorNPR-C, natriuretic peptide receptor CSAN, sinoatrial nodeSBP, systolic blood pressureV1/2(act), voltage for 50 channel activationcSNRT, corrected sinoatrial node recovery timefibrosishypertensionion currentsnatriuretic peptidesinoatrial node
DOI
10.1016/j.jacbts.2018.08.004
PMID
30623142
发布时间
2020-10-01
- 浏览14
JACC. Basic to translational science
2018年3卷6期
824-843页
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