IL-17A Recruits Rab35 to IL-17R to Mediate PKCα-Dependent Stress Fiber Formation and Airway Smooth Muscle Contractility.

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作者： Katarzyna,Bulek ^[1] ; Xing,Chen ^[2] ; Vandy,Parron ^[3] ; Aparna,Sundaram ^[4] ; Tomasz,Herjan ^[5] ; Suidong,Ouyang ^[3] ; Caini,Liu ^[6] ; Alana,Majors ^[3] ; Jarod,Zepp ^[3] ; Ji,Gao ^[3] ; Ashok,Dongre ^[3] ; Malgorzata,Bodaszewska-Lubas ^[7] ; Arnaud,Echard ^[7] ; Mark,Aronica ^[2] ; Julie,Carman ^[8] ; Stavros,Garantziotis ^[3] ; Dean,Sheppard ^[7] ; Xiaoxia,Li ^[4]

作者单位： Department of Inflammation and Immunity, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, OH 44195; bulekk@ccf.org lix@ccf.org. ^[1] Department of Immunology, Faculty of Biochemistry, Biophysics, and Biotechnology, Jagiellonian University, 30-387 Krakow, Poland. ^[2] Department of Inflammation and Immunity, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, OH 44195. ^[3] Division of Intramural Research, National Institute of Environmental Health Sciences, Research Triangle Park, NC 27709. ^[4] Lung Biology Center, University of California San Francisco, San Francisco, CA 94143. ^[5] Department of General Biochemistry, Faculty of Biochemistry, Biophysics and Biotechnology, Jagiellonian University, 30-387 Krakow, Poland. ^[6] Discovery Biology, Bristol-Myers Squibb, Princeton, NJ 08543; and. ^[7] Membrane Traffic and Cell Division Lab, Cell Biology and Infection Department, Pasteur Institute, 75015 Paris, France. ^[8]