Ferritinophagy activation and sideroflexin1-dependent mitochondria iron overload is involved in apelin-13-induced cardiomyocytes hypertrophy.

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作者： Mingzhu,Tang ^[1] ; Zhen,Huang ^[1] ; Xuling,Luo ^[1] ; Meiqing,Liu ^[1] ; Lingzhi,Wang ^[1] ; Zhihao,Qi ^[1] ; Shifang,Huang ^[1] ; Jiuchang,Zhong ^[2] ; Jian-Xiong,Chen ^[3] ; Lanfang,Li ^[4] ; Di,Wu ^[5] ; Linxi,Chen ^[6]

作者单位： Institute of Pharmacy and Pharmacology, Hunan Province Cooperative Innovation Center for Molecular Target New Drug Study, Learning Key Laboratory for Pharmacoproteomics, University of South China, Hengyang, 421001, China. ^[1] Heart Center and Beijing Key Laboratory of Hypertension, Beijing Chaoyang Hospital Affiliated to Capital Medical University, Beijing, 100020, China. ^[2] Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson, MS, USA. ^[3] Institute of Pharmacy and Pharmacology, Hunan Province Cooperative Innovation Center for Molecular Target New Drug Study, Learning Key Laboratory for Pharmacoproteomics, University of South China, Hengyang, 421001, China. Electronic address: llanfang6@126.com. ^[4] Institute of Pharmacy and Pharmacology, Hunan Province Cooperative Innovation Center for Molecular Target New Drug Study, Learning Key Laboratory for Pharmacoproteomics, University of South China, Hengyang, 421001, China. Electronic address: 15574737843@163.com. ^[5] Institute of Pharmacy and Pharmacology, Hunan Province Cooperative Innovation Center for Molecular Target New Drug Study, Learning Key Laboratory for Pharmacoproteomics, University of South China, Hengyang, 421001, China. Electronic address: lxchen6@126.com. ^[6]

关键词 Apelin-13 Cardiomyocytes hypertrophy Ferritin Ferritinophagy Mitochondria iron overload ROS sideroflexin1