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Cardiolipin-deficient cells depend on anaplerotic pathways to ameliorate defective TCA cycle function.

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第一作者： Vaishnavi,Raja

第一单位： Department of Biological Sciences, Wayne State University, Detroit, MI 48202, United States of America.

作者： Vaishnavi,Raja ^[1] ; Michael,Salsaa ^[1] ; Amit S,Joshi ^[1] ; Yiran,Li ^[1] ; Carlo W T,van Roermund ^[2] ; Nadia,Saadat ^[3] ; Pablo,Lazcano ^[1] ; Michael,Schmidtke ^[1] ; Maik,Hüttemann ^[4] ; Smiti V,Gupta ^[3] ; Ronald J A,Wanders ^[2] ; Miriam L,Greenberg ^[5]

作者单位： Department of Biological Sciences, Wayne State University, Detroit, MI 48202, United States of America. ^[1] Laboratory Genetic Metabolic Diseases, Department of Clinical Chemistry, Academic Medical Center, Amsterdam, the Netherlands. ^[2] Department of Nutrition and Food Science, Wayne State University, Detroit, MI 48202, United States of America. ^[3] Center for Molecular Medicine and Genetics, Wayne State University School of Medicine, Detroit, Michigan 48201. ^[4] Department of Biological Sciences, Wayne State University, Detroit, MI 48202, United States of America. Electronic address: mgreenberg@wayne.edu. ^[5]

关键词 Cardiolipin Carnitine shuttle Glyoxylate cycle RTG pathway TCA cycle β-Oxidation