ProNGF increases breast tumor aggressiveness through functional association of TrkA with EphA2.-论文-万方医学网

第一作者： Romain,Lévêque

第一单位： Inserm, U908, F-59000, Lille, France; Univ. Lille, U908 - CPAC, Cell Plasticity and Cancer, F-59000, Lille, France.

作者： Romain,Lévêque ^[1] ; Cyril,Corbet ^[1] ; Léo,Aubert ^[1] ; Matthieu,Guilbert ^[1] ; Chann,Lagadec ^[2] ; Eric,Adriaenssens ^[1] ; Jérémy,Duval ^[1] ; Pascal,Finetti ^[3] ; Daniel,Birnbaum ^[3] ; Nicolas,Magné ^[4] ; Valérie,Chopin ^[5] ; François,Bertucci ^[3] ; Xuefen,Le Bourhis ^[1] ; Robert-Alain,Toillon ^[6]

作者单位： Inserm, U908, F-59000, Lille, France; Univ. Lille, U908 - CPAC, Cell Plasticity and Cancer, F-59000, Lille, France. ^[1] Inserm, U908, F-59000, Lille, France. ^[2] Département D'Oncologie Moléculaire, Institut Paoli-Calmette, CRCM, UMR1068 Inserm, UMR7258 CNRS, Aix-Marseille Université, 13273, Marseille, France. ^[3] Département de Radiothérapie, Institut de Cancérologie Lucien Neuwirth, 42270, Saint Priest en Jarez, France; Radiobiologie Cellulaire et Moléculaire, EMR3738 - Equipe 4, Faculté de Médecine Lyon-Sud, 69000, Lyon, France. ^[4] Univ. Lille, U908 - CPAC, Cell Plasticity and Cancer, F-59000, Lille, France; Université de Picardie, 80000, Amiens, France. ^[5] Inserm, U908, F-59000, Lille, France; Univ. Lille, U908 - CPAC, Cell Plasticity and Cancer, F-59000, Lille, France. Electronic address: robert-alain.toillon@univ-lille.fr. ^[6]

医学主题词衔接蛋白质类, 膜泡运输(Adaptor Proteins, Vesicular Transport);动物(Animals);抗肿瘤药(Antineoplastic Agents);脑肿瘤(Brain Neoplasms);乳腺肿瘤(Breast Neoplasms);细胞增殖(Cell Proliferation);膜附着型配体A2(Ephrin-A2);女(雌)性(Female);人类(Humans);小鼠, SCID(Mice, SCID);神经生长因子(Nerve Growth Factor);磷酰化(Phosphorylation);蛋白质结合(Protein Binding);蛋白激酶抑制剂(Protein Kinase Inhibitors);蛋白质前体(Protein Precursors);受体, EphA2(Receptor, EphA2);受体, trkA(Receptor, trkA);信号传导(Signal Transduction);肿瘤负荷(Tumor Burden);异种移植模型抗肿瘤试验(Xenograft Model Antitumor Assays);src族激酶类(src-Family Kinases)

DOI 10.1016/j.canlet.2019.02.019

PMID 30771434

发布时间 2021-12-04

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ProNGF increases breast tumor aggressiveness through functional association of TrkA with EphA2.

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ProNGF increases breast tumor aggressiveness through functional association of TrkA with EphA2.

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