Dysfunctional high-density lipoprotein activates toll-like receptors via serum amyloid A in vascular smooth muscle cells.
第一作者:
Mirjam,Schuchardt
第一单位:
Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt Universität zu Berlin, and Berlin Institute of Health, Department of Nephrology, Hindenburgdamm 30, 12203, Berlin, Germany.
作者:
主题词
动物(Animals);细胞, 培养的(Cells, Cultured);趋化因子CCL2(Chemokine CCL2);人类(Humans);脂蛋白类, HDL(Lipoproteins, HDL);小鼠(Mice);肌, 平滑, 血管(Muscle, Smooth, Vascular);肌细胞, 平滑肌(Myocytes, Smooth Muscle);大鼠(Rats);血清淀粉样蛋白A(Serum Amyloid A Protein);Toll样受体2(Toll-Like Receptor 2);Toll样受体4(Toll-Like Receptor 4);Toll样受体(Toll-Like Receptors)
DOI
10.1038/s41598-019-39846-3
PMID
30833653
发布时间
2020-09-21
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