Myopia disease mouse models: a missense point mutation (S673G) and a protein-truncating mutation of the <i>Zfp644</i> mimic human disease phenotype.

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作者： Katarzyna I,Szczerkowska ^[1] ; Silvia,Petrezselyova ^[1] ; Jiri,Lindovsky ^[2] ; Marcela,Palkova ^[2] ; Jan,Dvorak ^[2] ; Peter,Makovicky ^[1] ; Mingyan,Fang ^[2] ; Chongyi,Jiang ^[3] ; Lingyan,Chen ^[4] ; Mingming,Shi ^[5] ; Xiao,Liu ^[4] ; Jianguo,Zhang ^[5] ; Agnieszka,Kubik-Zahorodna ^[4] ; Bjoern,Schuster ^[5] ; Inken M,Beck ^[4] ; Vendula,Novosadova ^[5] ; Jan,Prochazka ^[4] ; Radislav,Sedlacek ^[5]

作者单位： 1Laboratory of Transgenic Models of Diseases, Institute of Molecular Genetics CAS, Prumyslova 595, Vestec, 252 50 Prague, Czech Republic. ^[1] 2Czech Centre for Phenogenomics, Institute of Molecular Genetics CAS, Prague, Czech Republic. ^[2] 3Division of Clinical Immunology, Department of Laboratory Medicine, Karolinska Institutet at Karolinska University Hospital Huddinge, Stockholm, Sweden. ^[3] 4BGI-Shenzhen, Shenzhen, 518083 China. ^[4] 5China National GeneBank, BGI-Shenzhen, Shenzhen, 518120 China. ^[5] 6Animal Research Center, Ulm University, Ulm, Germany. ^[6]

关键词 Eye Genetics Mouse model Myopia Vision Zinc finger 644

DOI 10.1186/s13578-019-0280-4

PMID 30834109

发布时间 2020-10-01

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Cell & bioscience

2019年9卷

21页

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Myopia disease mouse models: a missense point mutation (S673G) and a protein-truncating mutation of the <i>Zfp644</i> mimic human disease phenotype.

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Myopia disease mouse models: a missense point mutation (S673G) and a protein-truncating mutation of the <i>Zfp644</i> mimic human disease phenotype.

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