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The Skin Commensal Yeast Malassezia Triggers a Type 17 Response that Coordinates Anti-fungal Immunity and Exacerbates Skin Inflammation.

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第一作者： Florian,Sparber

第一单位： Section of Immunology, Vetsuisse Faculty, University of Zürich, Zürich 8057, Switzerland.

作者： Florian,Sparber ^[1] ; Corinne,De Gregorio ^[2] ; Simone,Steckholzer ^[1] ; Filipa M,Ferreira ^[3] ; Tamas,Dolowschiak ^[4] ; Fiorella,Ruchti ^[1] ; Florian R,Kirchner ^[1] ; Sarah,Mertens ^[1] ; Immo,Prinz ^[5] ; Nicole,Joller ^[4] ; Thorsten,Buch ^[3] ; Martin,Glatz ^[6] ; Federica,Sallusto ^[7] ; Salomé,LeibundGut-Landmann ^[8]

作者单位： Section of Immunology, Vetsuisse Faculty, University of Zürich, Zürich 8057, Switzerland. ^[1] Institute for Research in Biomedicine, Università della Svizzera Italiana, Bellinzona 6500, Switzerland. ^[2] Institute of Laboratory Animal Science, University of Zürich, Schlieren 8952, Switzerland. ^[3] Institute of Experimental Immunology, University of Zürich, Zürich 8057, Switzerland. ^[4] Institute of Immunology, Hannover Medical School, Hannover 30625, Germany. ^[5] Department of Dermatology, University and University Hospital of Zürich, Zürich 8091, Switzerland. ^[6] Institute for Research in Biomedicine, Università della Svizzera Italiana, Bellinzona 6500, Switzerland; Institute of Microbiology, ETH Zürich, Zürich 8093, Switzerland. ^[7] Section of Immunology, Vetsuisse Faculty, University of Zürich, Zürich 8057, Switzerland. Electronic address: salome.leibundgut-landmann@uzh.ch. ^[8]

关键词 Malassezia Th17 atopic dermatitis fungal commensalism inflammation interleukin-17 interleukin-23 skin immunity skin microbiota γδ T?cells