Glycine Protects against Hypoxic-Ischemic Brain Injury by Regulating Mitochondria-Mediated Autophagy via the AMPK Pathway.

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第一作者： Chen-Chen,Cai

第一单位： Department of Neonatology, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325027, China.

作者： Chen-Chen,Cai ^[1] ; Jiang-Hu,Zhu ^[1] ; Li-Xia,Ye ^[1] ; Yuan-Yuan,Dai ^[1] ; Ming-Chu,Fang ^[1] ; Ying-Ying,Hu ^[1] ; Shu-Lin,Pan ^[1] ; Si,Chen ^[1] ; Pei-Jun,Li ^[1] ; Xiao-Qin,Fu ^[1] ; Zhen-Lang,Lin ^[1]

作者单位： Department of Neonatology, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325027, China. ^[1]

主题词动物(Animals);自噬(Autophagy);甘氨酸(Glycine);缺氧缺血, 脑(Hypoxia-Ischemia, Brain);预后(Prognosis);蛋白激酶类(Protein Kinases);大鼠(Rats)

DOI 10.1155/2019/4248529

PMID 30881590

发布时间 2021-12-04

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Oxidative medicine and cellular longevity

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Glycine Protects against Hypoxic-Ischemic Brain Injury by Regulating Mitochondria-Mediated Autophagy via the AMPK Pathway.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

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Glycine Protects against Hypoxic-Ischemic Brain Injury by Regulating Mitochondria-Mediated Autophagy via the AMPK Pathway.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

相关期刊

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