Calcium-dependent blood-brain barrier breakdown by NOX5 limits postreperfusion benefit in stroke.

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第一作者： Ana I,Casas

第一单位： Department of Pharmacology and Personalised Medicine, CARIM, Maastricht University, Maastricht, The Netherlands.

作者： Ana I,Casas ^[1] ; Pamela Wm,Kleikers ^[1] ; Eva,Geuss ^[2] ; Friederike,Langhauser ^[3] ; Thure,Adler ^[4] ; Dirk H,Busch ^[5] ; Valerie,Gailus-Durner ^[5] ; Martin Hrabê,de Angelis ^[4] ; Javier,Egea ^[4] ; Manuela G,Lopez ^[6] ; Christoph,Kleinschnitz ^[7] ; Harald Hhw,Schmidt ^[8]

作者单位： Department of Pharmacology and Personalised Medicine, CARIM, Maastricht University, Maastricht, The Netherlands. ^[1] Department of Neurology, University Hospital Würzburg, Würzburg, Germany. ^[2] Department of Neurology, University Clinics Essen, Essen, Germany. ^[3] German Mouse Clinic, Institute of Experimental Genetics, Helmholtz Zentrum München, German Research Center for Environmental Health, Neuherberg, Germany. ^[4] Institute for Medical Microbiology, Immunology and Hygiene, Technical University of Munich, Munich, Germany. ^[5] Chair of Experimental Genetics, School of Life Science Weihenstephan, Technical University of Munich, Freising, Germany. ^[6] German Center for Diabetes Research (DZD), Neuherberg, Germany. ^[7] Instituto de Investigación Sanitaria, Servicio de Farmacología Clínica, Hospital Universitario de la Princesa, Madrid, Spain. ^[8] Institute Teofilo Hernando for Drug Discovery, Department of Pharmacology, School of Medicine, Universidad Autónoma de Madrid, Madrid, Spain. ^[9]

关键词 Calcium Neuroscience Pharmacology Therapeutics hypoxia