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首页> Nucleic acids research> Deletion of Gtpbp3 in zebrafish revealed the hypertrophic cardiomyopathy manifested by aberrant mitochondrial tRNA metabolism.

Deletion of Gtpbp3 in zebrafish revealed the hypertrophic cardiomyopathy manifested by aberrant mitochondrial tRNA metabolism.

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作者： Danni,Chen ^[1] ; Zengming,Zhang ^[2] ; Chao,Chen ^[1] ; Shihao,Yao ^[2] ; Qingxian,Yang ^[1] ; Feng,Li ^[2] ; Xiao,He ^[1] ; Cheng,Ai ^[2] ; Meng,Wang ^[1] ; Min-Xin,Guan ^[2]

作者单位： Division of Medical Genetics and Genomics, The Children's Hospital, Zhejiang University School of Medicine, Hangzhou, Zhejiang 310058, China. ^[1] Institute of Genetics, Zhejiang University School of Medicine, Hangzhou, Zhejiang 310058, China. ^[2] Department of Human Genetics, Zhejiang University School of Medicine, Hangzhou, Zhejiang 310058, China. ^[3] Key Laboratory of Reproductive Genetics, Ministry of Education, Zhejiang University, Hangzhou, Zhejiang 310058, China. ^[4] Joint Institute of Genetics and Genome Medicine between Zhejiang University and University of Toronto, Hangzhou, Zhejiang 310058, China. ^[5]

主题词氨基酰化(Aminoacylation);动物(Animals);心肌病, 肥厚性(Cardiomyopathy, Hypertrophic);GTP结合蛋白质类(GTP-Binding Proteins);基因缺失(Gene Deletion);原位杂交(In Situ Hybridization);线粒体(Mitochondria);线粒体蛋白质类(Mitochondrial Proteins);突变(Mutation);肌细胞, 心脏(Myocytes, Cardiac);表型(Phenotype);蛋白质构象(Protein Conformation);RNA, 转移(RNA, Transfer);转基因(Transgenes);斑马鱼(Zebrafish);斑马鱼蛋白质类(Zebrafish Proteins)

DOI 10.1093/nar/gkz218

PMID 30916346

发布时间 2020-03-09

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Nucleic acids research

Nucleic acids research

2019年47卷10期

5341-5355页

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