Influence of genetic copy number variants of the human GLUT3 glucose transporter gene <i>SLC2A3</i> on protein expression, glycolysis and rheumatoid arthritis risk: A genetic replication study.

导出 LinkOut

翻译打印收藏纠错

作者： Kim R,Simpfendorfer ^[1] ; Wentian,Li ^[2] ; Andrew,Shih ^[1] ; Hongxiu,Wen ^[1] ; Harini P,Kothari ^[1] ; Edward A,Einsidler ^[1] ; Arthur,Wuster ^[1] ; Julie,Hunkapiller ^[3] ; Timothy W,Behrens ^[3] ; Robert R,Graham ^[3] ; Michael J,Townsend ^[3] ; Doron M,Behar ^[4] ; Rui,Hu ^[5] ; Elliott,Greenspan ^[5] ; Peter K,Gregersen ^[5]

作者单位： Robert S. Boas Center for Genomics and Human Genetics, the Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, New York, USA. ^[1] Department of Molecular Medicine, Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, New York, USA. ^[2] Department of Human Genetics, Genentech Inc., 1 DNA Way, South San Francisco, California, USA. ^[3] Department of Biomarker Discovery OMNI, Genentech Inc., 1 DNA Way, South San Francisco, California, USA. ^[4] Gene by Gene, Genomic Research Center, Houston, TX, USA. ^[5]

关键词 Copy number variant Deletion GLUT3 Glucose transport Glycolysis Rheumatoid arthritis SLC2A3

DOI 10.1016/j.ymgmr.2019.100470

PMID 30997344

发布时间 2022-04-08

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览8

Molecular genetics and metabolism reports

2019年19卷

100470页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Influence of genetic copy number variants of the human GLUT3 glucose transporter gene <i>SLC2A3</i> on protein expression, glycolysis and rheumatoid arthritis risk: A genetic replication study.

Molecular genetics and metabolism reports

相似文献

Molecular genetics and metabolism reports

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Influence of genetic copy number variants of the human GLUT3 glucose transporter gene <i>SLC2A3</i> on protein expression, glycolysis and rheumatoid arthritis risk: A genetic replication study.

Molecular genetics and metabolism reports

相似文献

Molecular genetics and metabolism reports

相关期刊

检索历史清除