eIF4B and eIF4H mediate GR production from expanded G4C2 in a Drosophila model for C9orf72-associated ALS.

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第一作者： Lindsey D,Goodman

第一单位： Neuroscience Graduate Group, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, 19104, USA.

作者： Lindsey D,Goodman ^[1] ; Mercedes,Prudencio ^[2] ; Ananth R,Srinivasan ^[3] ; Olivia M,Rifai ^[3] ; Virginia M-Y,Lee ^[4] ; Leonard,Petrucelli ^[2] ; Nancy M,Bonini ^[5]

作者单位： Neuroscience Graduate Group, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, 19104, USA. ^[1] Department of Neuroscience, Mayo Clinic, Jacksonville, FL, 32224, USA. ^[2] Department of Biology, University of Pennsylvania, Philadelphia, PA, 19104, USA. ^[3] Center for Neurodegenerative Disease Research, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, 19104, USA. ^[4] Neuroscience Graduate Group, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, 19104, USA. nbonini@sas.upenn.edu.;Department of Biology, University of Pennsylvania, Philadelphia, PA, 19104, USA. nbonini@sas.upenn.edu. ^[5]

关键词 Amyotrophic lateral sclerosis (ALS) (Lou Gehrig disease)C9orf72 Drosophila Neurodegeneration RAN-translation eIF4B eIF4H

主题词肌萎缩侧索硬化(Amyotrophic Lateral Sclerosis);动物(Animals);动物, 基因修饰(Animals, Genetically Modified);DNA重复序列扩增(DNA Repeat Expansion);二肽类(Dipeptides);疾病模型, 动物(Disease Models, Animal);果蝇属(Drosophila);真核细胞起始因子类(Eukaryotic Initiation Factors);女(雌)性(Female);基因表达调控(Gene Expression Regulation);人类(Humans);男(雄)性(Male);RNA(RNA)

DOI 10.1186/s40478-019-0711-9

PMID 31023341

发布时间 2023-01-07

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Acta neuropathologica communications

2019年7卷1期

62页

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eIF4B and eIF4H mediate GR production from expanded G4C2 in a Drosophila model for C9orf72-associated ALS.

Acta neuropathologica communications

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Acta neuropathologica communications

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eIF4B and eIF4H mediate GR production from expanded G4C2 in a Drosophila model for C9orf72-associated ALS.

Acta neuropathologica communications

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Acta neuropathologica communications

相关期刊

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