Interleukin-1 receptor associated kinase (IRAK)-M -mediated type 2 microglia polarization ameliorates the severity of experimental autoimmune encephalomyelitis (EAE).

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第一作者： Baozhu,Liu

第一单位： Department of Neurology, Nanfang Hospital, Southern Medical University, Guangzhou, China.

作者： Baozhu,Liu ^[1] ; Yong,Gu ^[2] ; Shanshan,Pei ^[1] ; Yu,Peng ^[1] ; Jinyu,Chen ^[1] ; Lan V,Pham ^[3] ; Hai-Ying,Shen ^[4] ; Jun,Zhang ^[5] ; Honghao,Wang ^[6]

作者单位： Department of Neurology, Nanfang Hospital, Southern Medical University, Guangzhou, China. ^[1] Department of Encephalopathy, Hainan Province Hospital of Traditional Chinese Medicine, Haikou, China. ^[2] Department of Hematopathology, University of Texas, MD Anderson Cancer Center, USA. Electronic address: Lvpham@mdanderson.org. ^[3] Department of Neurobiology, Legacy Research Institute, 1225 NE 2nd Ave, Portland, OR, 97232, USA. Electronic address: hshen@downeurobiology.org. ^[4] Department of Surgical Oncology, University of Texas, MD Anderson Cancer Center, USA. Electronic address: jzhang32@mdanderson.org. ^[5] Department of Neurology, Nanfang Hospital, Southern Medical University, Guangzhou, China. Electronic address: wang_whh@163.com. ^[6]

关键词 Experimental autoimmune encephalomyelitis IRAK-M Microglia Multiple sclerosis TLR4