The Checkpoint Regulator SLAMF3 Preferentially Prevents Expansion of Auto-Reactive B Cells Generated by Graft-vs.-Host Disease.

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作者： Ninghai,Wang ^[1] ; Burcu,Yigit ^[1] ; Cees E,van der Poel ^[2] ; Marta,Cuenca ^[3] ; Michael C,Carroll ^[2] ; Roland W,Herzog ^[3] ; Pablo,Engel ^[4] ; Cox,Terhorst ^[1]

作者单位： Division of Immunology, Harvard Medical School, Beth Israel Deaconess Medical Center, Boston, MA, United States. ^[1] Program in Cellular and Molecular Medicine, Harvard Medical School, Boston Children's Hospital, Boston, MA, United States. ^[2] Herman B Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN, United States. ^[3] Immunology Unit, Department of Cell Biology, Immunology and Neurosciences, Medical School, University of Barcelona, Barcelona, Spain. ^[4]

关键词 B cells SLAMF3 alloimmunity autoreactive cGVHD

主题词动物(Animals);B淋巴细胞亚群(B-Lymphocyte Subsets);细胞增殖(Cell Proliferation);女(雌)性(Female);生发中心(Germinal Center);移植物抗宿主病(Graft vs Host Disease);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);信号传导(Signal Transduction);T淋巴细胞, 调节性(T-Lymphocytes, Regulatory)

DOI 10.3389/fimmu.2019.00831

PMID 31057553

发布时间 2020-09-24

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Frontiers in immunology

2019年10卷

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The Checkpoint Regulator SLAMF3 Preferentially Prevents Expansion of Auto-Reactive B Cells Generated by Graft-vs.-Host Disease.

Frontiers in immunology

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Frontiers in immunology

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The Checkpoint Regulator SLAMF3 Preferentially Prevents Expansion of Auto-Reactive B Cells Generated by Graft-vs.-Host Disease.

Frontiers in immunology

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Frontiers in immunology

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