Dynamic UTR Usage Regulates Alternative Translation to Modulate Gap Junction Formation during Stress and Aging.

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第一作者： Michael J,Zeitz

第一单位： Fralin Biomedical Research Institute, Roanoke, VA 24016, USA.

作者： Michael J,Zeitz ^[1] ; Patrick J,Calhoun ^[2] ; Carissa C,James ^[3] ; Thomas,Taetzsch ^[1] ; Kijana K,George ^[3] ; Stefanie,Robel ^[4] ; Gregorio,Valdez ^[5] ; James W,Smyth ^[6]

作者单位： Fralin Biomedical Research Institute, Roanoke, VA 24016, USA. ^[1] Fralin Biomedical Research Institute, Roanoke, VA 24016, USA; Department of Biological Sciences, Virginia State University and Polytechnic Institute, Blacksburg, VA 24060, USA. ^[2] Fralin Biomedical Research Institute, Roanoke, VA 24016, USA; Graduate Program in Translational Biology, Medicine, and Health, Virginia State University and Polytechnic Institute, Blacksburg, VA 24060, USA. ^[3] Fralin Biomedical Research Institute, Roanoke, VA 24016, USA; Department of Biological Sciences, Virginia State University and Polytechnic Institute, Blacksburg, VA 24060, USA; Virginia Tech Carilion School of Medicine, Roanoke, VA 24016, USA; School of Neuroscience, Virginia State University and Polytechnic Institute, Blacksburg, VA 24060, USA. ^[4] Fralin Biomedical Research Institute, Roanoke, VA 24016, USA; Department of Biological Sciences, Virginia State University and Polytechnic Institute, Blacksburg, VA 24060, USA; Virginia Tech Carilion School of Medicine, Roanoke, VA 24016, USA. ^[5] Fralin Biomedical Research Institute, Roanoke, VA 24016, USA; Department of Biological Sciences, Virginia State University and Polytechnic Institute, Blacksburg, VA 24060, USA; Virginia Tech Carilion School of Medicine, Roanoke, VA 24016, USA. Electronic address: smythj@vtc.vt.edu. ^[6]

关键词 TGF-β UTR aging connexin gap junction mRNA translation