Proteasome-dependent degradation of intracellular carbamylated proteins.

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作者： Aurore,Desmons ^[1] ; Anaïs,Okwieka ^[2] ; Manon,Doué ^[1] ; Laëtitia,Gorisse ^[1] ; Vincent,Vuiblet ^[1] ; Christine,Pietrement ^[3] ; Philippe,Gillery ^[4] ; Stéphane,Jaisson ^[1]

作者单位： Laboratory of Biochemistry and Molecular Biology, CNRS/URCA UMR N° 7369 MEDyC, Faculty of Medicine, University of Reims Champagne-Ardenne, Reims, France. ^[1] Laboratory of Pediatric Biology and Research, University Hospital of Reims, Reims, France. ^[2] Present address: Department of Laboratory Medicine, National Institutes of Health, Bethesda, MD 20892, USA. ^[3] Laboratory of Biopathology, University Hospital of Reims, Reims, France. ^[4] Department of Pediatrics (Nephrology unit), University Hospital of Reims, Reims, France. ^[5]

关键词 carbamylation cell aging homocitrulline nonenzymatic post-translational modifications proteasome proteostasis

主题词氰酸盐类(Cyanates);成纤维细胞(Fibroblasts);人类(Humans);蛋白酶体内肽酶复合物(Proteasome Endopeptidase Complex);皮肤(Skin);尿素(Urea)

DOI 10.18632/aging.102002

PMID 31170093

发布时间 2024-07-17

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