Exogenous H2S reduces the acetylation levels of mitochondrial respiratory enzymes via regulating the NAD+-SIRT3 pathway in cardiac tissues of db/db mice.

导出 OA

翻译打印收藏纠错

作者： Yu,Sun ^[1] ; Zongyan,Teng ^[2] ; Xiaojiao,Sun ^[1] ; Linxue,Zhang ^[1] ; Jian,Chen ^[1] ; Bingzhu,Wang ^[1] ; Fangping,Lu ^[1] ; Ning,Liu ^[1] ; Miao,Yu ^[1] ; Shuo,Peng ^[1] ; Yan,Wang ^[3] ; Dechao,Zhao ^[4] ; Yajun,Zhao ^[1] ; Huan,Ren ^[5] ; Zhongyi,Cheng ^[6] ; Shiyun,Dong ^[1] ; Fanghao,Lu ^[1] ; Weihua,Zhang ^[1]

作者单位： Department of Pathophysiology, Harbin Medical University , Harbin , China. ^[1] Department of Geriatrics, Second Affiliated Hospital of Harbin Medical University , Harbin , China. ^[2] Department of Urologic Surgery, First Affiliated Hospital of Harbin Medical University , Harbin , China. ^[3] Department of Cardiology, First affiliated hospital of Harbin Medical University , Harbin , China. ^[4] Department of Immunology, Harbin Medical University , Harbin , China. ^[5] Jingjie PTM BioLab, Co., Ltd. (Hangzhou) , Hangzhou , China. ^[6] Key Laboratory of Cardiovascular Medicine Research (Harbin Medical University), Ministry of Education , Harbin , China. ^[7]

关键词 SIRT3 hydrogen sulfide lysine acetylation mitochondrial respiratory chain type 2 diabetes