Thiamine and benfotiamine protect neuroblastoma cells against paraquat and β-amyloid toxicity by a coenzyme-independent mechanism.

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第一作者： Margaux,Sambon

第一单位： Laboratory of Neurophysiology, GIGA-Neurosciences, University of Liège, Liège, Belgium.

作者： Margaux,Sambon ^[1] ; Aurore,Napp ^[2] ; Alice,Demelenne ^[2] ; Julie,Vignisse ^[1] ; Pierre,Wins ^[1] ; Marianne,Fillet ^[2] ; Lucien,Bettendorff ^[1]

作者单位： Laboratory of Neurophysiology, GIGA-Neurosciences, University of Liège, Liège, Belgium. ^[1] Laboratory for the Analysis of Medicines, CIRM, Department of Pharmacy, University of Liège, Liège, Belgium. ^[2]

关键词 ARE, antioxidant response element BFT, benfotiamine Cell biology FBS, fetal bovine serum Neuroscience O-BT, O-benzoylthiamine PQ, paraquat ROS, reactive oxygen species S-BT, S-benzoylthiamine SuBT, sulbutiamine TPK, thiamine pyrophosphokinase ThDP, thiamine diphosphate ThMP, thiamine monophosphate

DOI 10.1016/j.heliyon.2019.e01710

PMID 31193162

发布时间 2022-04-08

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2019年5卷5期

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Thiamine and benfotiamine protect neuroblastoma cells against paraquat and β-amyloid toxicity by a coenzyme-independent mechanism.

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Thiamine and benfotiamine protect neuroblastoma cells against paraquat and β-amyloid toxicity by a coenzyme-independent mechanism.

Heliyon

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Heliyon

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