Particulate matter disrupts airway epithelial barrier via oxidative stress to promote <i>Pseudomonas aeruginosa</i> infection.

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第一作者： Jinguo,Liu

第一单位： Department of Pulmonary Medicine, Zhongshan Hospital, Fudan University and Shanghai Respiratory Research Institute, Shanghai 200032, China.

作者： Jinguo,Liu ^[1] ; Xiaoyan,Chen ^[1] ; Maosen,Dou ^[2] ; Hong,He ^[3] ; Mohan,Ju ^[4] ; Shimeng,Ji ^[1] ; Jian,Zhou ^[1] ; Cuicui,Chen ^[1] ; Donghui,Zhang ^[1] ; Changhong,Miao ^[1] ; Yuanlin,Song ^[3]

作者单位： Department of Pulmonary Medicine, Zhongshan Hospital, Fudan University and Shanghai Respiratory Research Institute, Shanghai 200032, China. ^[1] Department of Infectious Diseases, Zhongshan Hospital, Fudan University, Shanghai 200032, China. ^[2] Department of Anesthesiology, Fudan University Shanghai Cancer Center, Shanghai 200032, China. ^[3] Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, China. ^[4] Department of Respiratory Medicine, Shanghai Public Health Clinical Center, Fudan University, Shanghai 200032, China. ^[5] Department of Pulmonary Medicine, Zhongshan Hospital, Qingpu Branch, Shanghai 200032, China. ^[6]

关键词 BEAS-2B cells Particulate matter (PM)Pseudomonas aeruginosa tight junctions (TJs)

DOI 10.21037/jtd.2019.05.77

PMID 31372298

发布时间 2022-04-09

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