Mitofusin 2 Is Essential for IP<sub>3</sub>-Mediated SR/Mitochondria Metabolic Feedback in Ventricular Myocytes.

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作者： Lea K,Seidlmayer ^[1] ; Christine,Mages ^[2] ; Annette,Berbner ^[1] ; Petra,Eder-Negrin ^[2] ; Paula Anahi,Arias-Loza ^[2] ; Mathias,Kaspar ^[2] ; Moshi,Song ^[2] ; Gerald W,Dorn ^[2] ; Michael,Kohlhaas ^[3] ; Stefan,Frantz ^[3] ; Christoph,Maack ^[2] ; Brenda,Gerull ^[1] ; Elena N,Dedkova ^[2]

作者单位： Department of Internal Medicine, Cardiology, University Hospital Würzburg, Würzburg, Germany. ^[1] Comprehensive Heart Failure Center, University of Würzburg, Würzburg, Germany. ^[2] Department of Internal Medicine, Center for Pharmacogenomics, Washington University School of Medicine, St. Louis, MO, United States. ^[3] Department of Pharmacology, School of Medicine, University of California, Davis, Davis, CA, United States. ^[4] Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, Davis, CA, United States. ^[5]

关键词 ATP generation IP3 signaling Mfn2 KO mice SR/mitochondria metabolic feedback endothelin-1 mitochondrial mRyR1 mitofusin 2

DOI 10.3389/fphys.2019.00733

PMID 31379586

发布时间 2020-10-01

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Frontiers in physiology

2019年10卷

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Mitofusin 2 Is Essential for IP<sub>3</sub>-Mediated SR/Mitochondria Metabolic Feedback in Ventricular Myocytes.

Frontiers in physiology

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Mitofusin 2 Is Essential for IP<sub>3</sub>-Mediated SR/Mitochondria Metabolic Feedback in Ventricular Myocytes.

Frontiers in physiology

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Frontiers in physiology

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