RAD51AP1 Is an Essential Mediator of Alternative Lengthening of Telomeres.

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作者： Jonathan,Barroso-González ^[1] ; Laura,García-Expósito ^[1] ; Song My,Hoang ^[1] ; Michelle L,Lynskey ^[1] ; Justin L,Roncaioli ^[1] ; Arundhati,Ghosh ^[2] ; Callen T,Wallace ^[3] ; Marco,de Vitis ^[4] ; Mauro,Modesti ^[5] ; Kara A,Bernstein ^[2] ; Saumendra N,Sarkar ^[2] ; Simon C,Watkins ^[3] ; Roderick J,O'Sullivan ^[6]

作者单位： Department of Pharmacology and Chemical Biology, UPMC Hillman Cancer Center, University of Pittsburgh, School of Medicine, Pittsburgh, PA, USA. ^[1] Department of Microbiology and Molecular Genetics, UPMC Hillman Cancer Center, University of Pittsburgh, School of Medicine, Pittsburgh, PA, USA. ^[2] Department of Cell Biology, UPMC Hillman Cancer Center, University of Pittsburgh, School of Medicine, Pittsburgh, PA, USA. ^[3] Department of Science, University of Rome "ROMA TRE", 00146 Rome, Italy. ^[4] Cancer Research Center of Marseille, CNRS UMR7258, Inserm UMR1068, Aix Marseille Université U105; Institut Paoli Calmettes, 27 Boulevard Lei Roure CS30059, 13273 Marseille, Cedex 09, France. ^[5] Department of Pharmacology and Chemical Biology, UPMC Hillman Cancer Center, University of Pittsburgh, School of Medicine, Pittsburgh, PA, USA. Electronic address: osullivanr@upmc.edu. ^[6]

关键词 RAD51AP1 SUMOylation autophagy cancer homology-directed repair telomere