MPTP-induced dopaminergic neurotoxicity in mouse brain is attenuated after subsequent intranasal administration of (R)-ketamine: a role of TrkB signaling.
作者:
主题词
1-甲基-4-苯基-1,2,3,6-四氢吡啶(1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine);投药, 鼻内(Administration, Intranasal);麻醉药, 离解(Anesthetics, Dissociative);动物(Animals);脑(Brain);纹状体(Corpus Striatum);疾病模型, 动物(Disease Models, Animal);多巴胺(Dopamine);多巴胺质膜转运蛋白质类(Dopamine Plasma Membrane Transport Proteins);氯胺酮(Ketamine);男(雄)性(Male);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);新纹状体(Neostriatum);神经保护药(Neuroprotective Agents);帕金森病(Parkinson Disease);蛋白酪氨酸激酶类(Protein-Tyrosine Kinases);受体, trkB(Receptor, trkB);信号传导(Signal Transduction);黑质(Substantia Nigra);酪氨酸单氧化酶(Tyrosine 3-Monooxygenase)
DOI
10.1007/s00213-019-05346-5
PMID
31418048
发布时间
2020-06-04
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Psychopharmacology
83-92页
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