Sarm1 deletion reduces axon damage, demyelination, and white matter atrophy after experimental traumatic brain injury.

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作者： Christina M,Marion ^[1] ; Dennis P,McDaniel ^[2] ; Regina C,Armstrong ^[3]

作者单位： Center for Neuroscience and Regenerative Medicine, F. Edward Hebert School of Medicine, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA; Program in Neuroscience, F. Edward Hebert School of Medicine, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA. ^[1] Biomedical Instrumentation Center, F. Edward Hebert School of Medicine, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA; Department of Microbiology and Immunology, F. Edward Hebert School of Medicine, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA. ^[2] Center for Neuroscience and Regenerative Medicine, F. Edward Hebert School of Medicine, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA; Department of Anatomy, Physiology and Genetics, F. Edward Hebert School of Medicine, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA; Program in Neuroscience, F. Edward Hebert School of Medicine, Uniformed Services University of the Health Sciences, Bethesda, MD 20814, USA. Electronic address: regina.armstrong@usuhs.edu. ^[3]

关键词 Axon Corpus callosum Demyelination Myelin Myelin outfoldings SARM1 Sterile alpha and TIR containing motif 1 TBI Traumatic axonal injury White matter