换一批A bigenic mouse model of FSGS reveals perturbed pathways in podocytes, mesangial cells and endothelial cells.
第一作者：
Andrew S,Potter
第一单位：
Department of Pediatrics, Division of Developmental Biology, Cincinnati Children's Medical Center, Cincinnati, OH, United States of America.
作者：
医学主题词
衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);动物(Animals);细胞支架蛋白质类(Cytoskeletal Proteins);疾病模型, 动物(Disease Models, Animal);内皮细胞(Endothelial Cells);肾小球硬化症, 局灶节段性(Glomerulosclerosis, Focal Segmental);肾小球系膜细胞(Mesangial Cells);小鼠(Mice);分子靶向治疗(Molecular Targeted Therapy);突变(Mutation);表型(Phenotype);足细胞(Podocytes);原癌基因蛋白质c-fyn(Proto-Oncogene Proteins c-fyn)
DOI
10.1371/journal.pone.0216261
PMID
31461442
发布时间
2020-07-01
- 浏览1
PloS one
e0216261页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
