Delineating the role of FANCA in glucose-stimulated insulin secretion in β cells through its protein interactome.

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作者： Dragana,Lagundžin ^[1] ; Wen-Feng,Hu ^[2] ; Henry C H,Law ^[1] ; Kimiko L,Krieger ^[1] ; Fangfang,Qiao ^[1] ; Emalie J,Clement ^[1] ; Andjela T,Drincic ^[1] ; Olgica,Nedić ^[3] ; Michael J,Naldrett ^[4] ; Sophie,Alvarez ^[5] ; Nicholas T,Woods ^[5]

作者单位： Eppley Institute for Research in Cancer and Allied Diseases, Fred & Pamela Buffett Cancer Center, University of Nebraska Medical Center, Omaha, Nebraska, United States of America. ^[1] Mass Spectrometry and Proteomics Core Facility, University of Nebraska Medical Center, Omaha, Nebraska, United States of America. ^[2] Department of Internal Medicine: Diabetes, Endocrinology and Metabolism, University of Nebraska Medical Center, Omaha, Nebraska, United States of America. ^[3] Institute for the Application of Nuclear Energy, University of Belgrade, Banatska, Belgrade, Serbia. ^[4] Proteomics & Metabolomics Facility, Nebraska Center for Biotechnology, University of Nebraska-Lincoln, Nebraska, United States of America. ^[5]

主题词细胞系(Cell Line);DNA损伤(DNA Damage);范可尼贫血互补群蛋白质A(Fanconi Anemia Complementation Group A Protein);葡萄糖(Glucose);人类(Humans);胰岛素分泌细胞(Insulin-Secreting Cells)

DOI 10.1371/journal.pone.0220568

PMID 31461451

发布时间 2020-03-03

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