Insulin Receptor Isoform A Modulates Metabolic Reprogramming of Breast Cancer Cells in Response to IGF2 and Insulin Stimulation.

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第一作者： Veronica,Vella

第一单位： Endocrinology, Department of Clinical and Experimental Medicine, University of Catania, Garibaldi-Nesima Hospital, Catania 95122, Italy.

作者： Veronica,Vella ^[1] ; Maria Luisa,Nicolosi ^[1] ; Marika,Giuliano ^[1] ; Andrea,Morrione ^[2] ; Roberta,Malaguarnera ^[3] ; Antonino,Belfiore ^[4]

作者单位： Endocrinology, Department of Clinical and Experimental Medicine, University of Catania, Garibaldi-Nesima Hospital, Catania 95122, Italy. ^[1] Department of Pathology, Anatomy and Cell Biology, Thomas Jefferson University, Philadelphia, PA 19107, USA. ^[2] School of Human and Social Sciences, "Kore" University of Enna, Enna 94100, Italy. ^[3] Endocrinology, Department of Clinical and Experimental Medicine, University of Catania, Garibaldi-Nesima Hospital, Catania 95122, Italy. antonino.belfiore@unict.it. ^[4]

关键词 IGF1R IGF2 aerobic glycolysis breast cancer insulin receptor isoform A metabolic flexibility metabolic reprogramming

主题词抗代谢药(Antimetabolites);乳腺肿瘤(Breast Neoplasms);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);脱氧葡萄糖(Deoxyglucose);女(雌)性(Female);糖酵解(Glycolysis);人类(Humans);胰岛素(Insulin);胰岛素样生长因子Ⅱ(Insulin-Like Growth Factor II);二甲双胍(Metformin);线粒体(Mitochondria);蛋白质亚型(Protein Isoforms);受体, IGF1型(Receptor, IGF Type 1)

DOI 10.3390/cells8091017

PMID 31480557

发布时间 2020-05-14

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Insulin Receptor Isoform A Modulates Metabolic Reprogramming of Breast Cancer Cells in Response to IGF2 and Insulin Stimulation.

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Insulin Receptor Isoform A Modulates Metabolic Reprogramming of Breast Cancer Cells in Response to IGF2 and Insulin Stimulation.

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