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Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation.

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第一作者： Victoria E,Wang

第一单位： Department of Medicine, University of California, San Francisco, San Francisco, California.;Helen Diller Comprehensive Cancer Center, University of California, San Francisco, San Francisco, California.

作者： Victoria E,Wang ^[1] ; Jenny Y,Xue ^[2] ; Dennie T,Frederick ^[3] ; Yi,Cao ^[4] ; Eva,Lin ^[4] ; Catherine,Wilson ^[4] ; Anatoly,Urisman ^[5] ; David P,Carbone ^[6] ; Keith T,Flaherty ^[3] ; Rene,Bernards ^[6] ; Piro,Lito ^[2] ; Jeff,Settleman ^[7] ; Frank,McCormick ^[8]

作者单位： Department of Medicine, University of California, San Francisco, San Francisco, California.;Helen Diller Comprehensive Cancer Center, University of California, San Francisco, San Francisco, California. ^[1] Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, New York.;Weill Cornell/Rockefeller/Sloan Kettering Tri-Institutional MD-PhD Program, New York, New York.;Weill Cornell Medical College, Cornell University, New York, New York. ^[2] Massachusetts General Hospital Cancer Center, Boston, Massachusetts. ^[3] Discovery Oncology, Genentech, South San Francisco, California. ^[4] Helen Diller Comprehensive Cancer Center, University of California, San Francisco, San Francisco, California.;The Ohio State University Comprehensive Cancer Center, Columbus, Ohio. ^[5] Department of Molecular Carcinogenesis, Netherlands Cancer Institute, Amsterdam, the Netherlands. ^[6] Discovery Oncology, Genentech, South San Francisco, California. Frank.mccormick@ucsf.edu jeffrey.settleman@pfizer.com. ^[7] Helen Diller Comprehensive Cancer Center, University of California, San Francisco, San Francisco, California. Frank.mccormick@ucsf.edu jeffrey.settleman@pfizer.com. ^[8]