换一批TMEM158 promotes pancreatic cancer aggressiveness by activation of TGFβ1 and PI3K/AKT signaling pathway.
第一作者：
Yue,Fu
第一单位：
Department of General Surgery, The Affiliated Changzhou No. 2 People's Hospital of Nanjing Medical University, Changzhou, Jiangsu, China.
作者：
医学主题词
动物(Animals);细胞系, 肿瘤(Cell Line, Tumor);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);疾病恶化(Disease Progression);上皮-间质转化(Epithelial-Mesenchymal Transition);女(雌)性(Female);基因表达谱(Gene Expression Profiling);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);男(雄)性(Male);膜蛋白质类(Membrane Proteins);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);小鼠, 裸(Mice, Nude);中年人(Middle Aged);肿瘤侵润(Neoplasm Invasiveness);肿瘤移植(Neoplasm Transplantation);胰腺肿瘤(Pancreatic Neoplasms);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction);转化生长因子β1(Transforming Growth Factor beta1);移植, 异种(Transplantation, Heterologous);肿瘤抑制蛋白质类(Tumor Suppressor Proteins)
DOI
10.1002/jcp.29181
PMID
31531884
发布时间
2020-12-16
- 浏览2
Journal of cellular physiology
2761-2775页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
