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TRPC3 deficiency attenuates high salt-induced cardiac hypertrophy by alleviating cardiac mitochondrial dysfunction.

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第一作者： Tianyi,Ma

第一单位： Department of Hypertension and Endocrinology, Center for Hypertension and Metabolic Diseases, Daping Hospital, Third Military Medical University, Chongqing Institute of Hypertension, Chongqing, 400042, China.

作者： Tianyi,Ma ^[1] ; Shaoyang,Lin ^[1] ; Bin,Wang ^[1] ; Qianran,Wang ^[1] ; Weijie,Xia ^[1] ; Hexuan,Zhang ^[1] ; Yuanting,Cui ^[1] ; Chengkang,He ^[1] ; Hao,Wu ^[1] ; Fang,Sun ^[1] ; Zhigang,Zhao ^[1] ; Peng,Gao ^[1] ; Zhiming,Zhu ^[1] ; Daoyan,Liu ^[2]

作者单位： Department of Hypertension and Endocrinology, Center for Hypertension and Metabolic Diseases, Daping Hospital, Third Military Medical University, Chongqing Institute of Hypertension, Chongqing, 400042, China. ^[1] Department of Hypertension and Endocrinology, Center for Hypertension and Metabolic Diseases, Daping Hospital, Third Military Medical University, Chongqing Institute of Hypertension, Chongqing, 400042, China. Electronic address: daoyanliu@yahoo.com. ^[2]

关键词 Calcium homeostasis Cardiac hypertrophy High salt diet Mitochondria TRPC3