The three members of the Vav family proteins form complexes that concur to foam cell formation and atherosclerosis.

导出 OA

翻译打印收藏纠错

作者： Rong,Huang ^[1] ; Guo,Guo ^[2] ; Liaoxun,Lu ^[1] ; Rui,Fu ^[2] ; Jing,Luo ^[1] ; Zhuangzhuang,Liu ^[2] ; Yanrong,Gu ^[3] ; Wenyi,Yang ^[1] ; Qianqian,Zheng ^[2] ; Tianzhu,Chao ^[1] ; Le,He ^[2] ; Ying,Wang ^[3] ; Zhiguo,Niu ^[1] ; Hui,Wang ^[2] ; Toby,Lawrence ^[1] ; Marie,Malissen ^[2] ; Bernard,Malissen ^[1] ; Yinming,Liang ^[2] ; Lichen,Zhang ^[3]

作者单位： Laboratory of Genetic Regulators in the Immune System, Henan Collaborative Innovation Center of Molecular Diagnosis and Laboratory Medicine, Xinxiang Medical University, Henan Province, China. ^[1] Henan Key Laboratory of Immunology and Targeted Therapy, School of Laboratory Medicine, Xinxiang Medical University, Henan Province, China. ^[2] Institute of Psychiatry and Neuroscience, Xinxiang Medical University, Henan Province, China. ^[3] Centre for Inflammation Biology and Cancer Immunology, School of Immunology & Microbial Sciences, King's College London, London, United Kingdom. ^[4] Centre d'Immunologie de Marseille-Luminy, UM2 Aix-Marseille Université, Marseille, France. ^[5] INSERM U1104, Marseille, France. ^[6] Centre National de la Recherche Scientifique, Unité Mixte de Recherche 7280, Marseille, France. ^[7] Laboratory of Genetic Regulators in the Immune System, Henan Collaborative Innovation Center of Molecular Diagnosis and Laboratory Medicine, Xinxiang Medical University, Henan Province, China yinming.liang@foxmail.com zhanglichen@xxmu.edu.cn. ^[8]

关键词 CD36 Vav2 endocytosis low-density lipoprotein metabolism