Disrupting Myelin-Specific Th17 Cell Gut Homing Confers Protection in an Adoptive Transfer Experimental Autoimmune Encephalomyelitis.-论文-万方医学网

作者： Donovan,Duc ^[1] ; Solenne,Vigne ^[1] ; Jeremiah,Bernier-Latmani ^[2] ; Yannick,Yersin ^[1] ; Florian,Ruiz ^[1] ; Nadia,Gaïa ^[3] ; Stefano,Leo ^[3] ; Vladimir,Lazarevic ^[3] ; Jacques,Schrenzel ^[3] ; Tatiana V,Petrova ^[2] ; Caroline,Pot ^[4]

作者单位： Laboratories of Neuroimmunology, Neuroscience Research Center and Service of Neurology, Department of Clinical Neurosciences, Lausanne University Hospital and University of Lausanne, Chemin des Boveresses 155, 1066 Epalinges, Switzerland. ^[1] Department of Oncology, Lausanne University Hospital and University of Lausanne, and Ludwig Institute for Cancer Research Lausanne, Chemin des Boveresses 155, 1066 Epalinges, Switzerland. ^[2] Genomic Research Laboratory, Division of Infectious Diseases, Department of Medicine, Geneva University Hospitals and University of Geneva, Rue Gabrielle-Perret-Gentil 4, 1211 Geneva, Switzerland. ^[3] Laboratories of Neuroimmunology, Neuroscience Research Center and Service of Neurology, Department of Clinical Neurosciences, Lausanne University Hospital and University of Lausanne, Chemin des Boveresses 155, 1066 Epalinges, Switzerland. Electronic address: caroline.pot-kreis@chuv.ch. ^[4]

主题词过继转移(Adoptive Transfer);动物(Animals);抗菌药(Anti-Bacterial Agents);抗原(Antigens);细胞黏附分子(Cell Adhesion Molecules);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);中枢神经系统(Central Nervous System);结肠(Colon);疾病模型, 动物(Disease Models, Animal);疾病恶化(Disease Progression);脑脊髓炎, 自身免疫性, 实验性(Encephalomyelitis, Autoimmune, Experimental);整合素类(Integrins);配体(Ligands);淋巴管(Lymphatic Vessels);小鼠, 近交C57BL(Mice, Inbred C57BL);黏膜(Mucous Membrane);髓鞘(Myelin Sheath);蛋白质结合(Protein Binding);受体, 抗原, T细胞(Receptors, Antigen, T-Cell);Th17细胞(Th17 Cells)

DOI 10.1016/j.celrep.2019.09.002

PMID 31597098

发布时间 2020-09-08

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Disrupting Myelin-Specific Th17 Cell Gut Homing Confers Protection in an Adoptive Transfer Experimental Autoimmune Encephalomyelitis.

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Disrupting Myelin-Specific Th17 Cell Gut Homing Confers Protection in an Adoptive Transfer Experimental Autoimmune Encephalomyelitis.

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