Neural connectivity predicts spreading of alpha-synuclein pathology in fibril-injected mouse models: Involvement of retrograde and anterograde axonal propagation.
第一作者:
Christopher,Mezias
第一单位:
Weill Cornell Medicine of Cornell University, Department of Neuroscience, New York, NY 10065, USA; Weill Cornell Medicine of Cornell University, Department of Radiology, New York, NY 10065, USA. Electronic address: chm2062@med.cornell.edu.
作者:
主题词
动物(Animals);轴突运输(Axonal Transport);疾病模型, 动物(Disease Models, Animal);包涵体(Inclusion Bodies);小鼠(Mice);模型, 理论(Models, Theoretical);神经通路(Neural Pathways);神经元(Neurons);帕金森病(Parkinson Disease);α突触核蛋白(alpha-Synuclein)
DOI
10.1016/j.nbd.2019.104623
PMID
31628991
发布时间
2021-11-09
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Neurobiology of disease
104623页
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