<i>Shank3</i> Exons 14-16 Deletion in Glutamatergic Neurons Leads to Social and Repetitive Behavioral Deficits Associated With Increased Cortical Layer 2/3 Neuronal Excitability.

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作者： Taesun,Yoo ^[1] ; Heejin,Cho ^[1] ; Haram,Park ^[2] ; Jiseok,Lee ^[2] ; Eunjoon,Kim ^[1]

作者单位： Department of Biological Sciences, Korea Advanced Institute of Science and Technology (KAIST), Daejeon, South Korea. ^[1] Center for Synaptic Brain Dysfunctions, Institute for Basic Science (IBS), Daejeon, South Korea. ^[2]

关键词 Phelan-McDermid syndrome Shank3 autism medial prefrontal cortex repetitive behavior social interaction striatum

DOI 10.3389/fncel.2019.00458

PMID 31649512

发布时间 2020-09-30

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Frontiers in cellular neuroscience

2019年13卷

458页

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<i>Shank3</i> Exons 14-16 Deletion in Glutamatergic Neurons Leads to Social and Repetitive Behavioral Deficits Associated With Increased Cortical Layer 2/3 Neuronal Excitability.

Frontiers in cellular neuroscience

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Frontiers in cellular neuroscience

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<i>Shank3</i> Exons 14-16 Deletion in Glutamatergic Neurons Leads to Social and Repetitive Behavioral Deficits Associated With Increased Cortical Layer 2/3 Neuronal Excitability.

Frontiers in cellular neuroscience

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Frontiers in cellular neuroscience

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