Leptin Restores Endothelial Function via Endothelial PPARγ-Nox1-Mediated Mechanisms in a Mouse Model of Congenital Generalized Lipodystrophy.
第一作者:
Thiago,Bruder-Nascimento
第一单位:
From the Vascular Biology Center (T.B.-N., J.L.F., S.H., S.K., G.A., D.J.R.F., E.J.B.), Medical College of Georgia, Augusta University.;Department of Pediatrics, Division of Endocrinology, University of Pittsburgh, PA (T.B.-N.).
作者:
医学主题词
方差分析(Analysis of Variance);动物(Animals);疾病模型, 动物(Disease Models, Animal);内皮, 血管(Endothelium, Vascular);瘦素(Leptin);脂质营养不良, 先天性全身性(Lipodystrophy, Congenital Generalized);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);PPARγ(PPAR gamma);随机分配(Random Allocation);活性氧(Reactive Oxygen Species);参考值(Reference Values);治疗结果(Treatment Outcome)
DOI
10.1161/HYPERTENSIONAHA.119.13398
PMID
31656096
发布时间
2020-11-28
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Hypertension (Dallas, Tex.
1399-1408页
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