Innate immune molecule surfactant protein D attenuates sepsis-induced acute kidney injury through modulating apoptosis and NFκB-mediated inflammation.

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作者： Shi-Jun,Lu ^[1] ; Jian-Hua,Xu ^[1] ; Zhao-Feng,He ^[2] ; Peng,Wu ^[1] ; Chao,Ning ^[1] ; Hai-Yan,Li ^[1]

作者单位： Intensive Care Unit, Linyi Central Hospital, Linyi, Shandong, China. ^[1] Intensive Care Unit, Ju County People's Hospital, Rizhao, Shandong, China. ^[2]

关键词 NFκB acute kidney injury apoptosis sepsis surfactant protein-D

主题词急性肾损伤(Acute Kidney Injury);动物(Animals);细胞凋亡(Apoptosis);疾病模型, 动物(Disease Models, Animal);免疫, 先天(Immunity, Innate);炎症(Inflammation);男(雄)性(Male);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);NF-κB(NF-kappa B);肺表面活性物质相关蛋白质D(Pulmonary Surfactant-Associated Protein D);肺表面活性剂(Pulmonary Surfactants);脓毒症(Sepsis)

DOI 10.1111/iwj.13237

PMID 31701658

发布时间 2021-07-03

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International wound journal

2020年17卷1期

100-106页

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Innate immune molecule surfactant protein D attenuates sepsis-induced acute kidney injury through modulating apoptosis and NFκB-mediated inflammation.

International wound journal

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International wound journal

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Innate immune molecule surfactant protein D attenuates sepsis-induced acute kidney injury through modulating apoptosis and NFκB-mediated inflammation.

International wound journal

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International wound journal

相关期刊

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