Astragaloside-IV protects H9C2(2-1) cardiomyocytes from high glucose-induced injury via miR-34a-mediated autophagy pathway.

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作者： Yaobin,Zhu ^[1] ; Xin,Qian ^[2] ; Jingjing,Li ^[2] ; Xing,Lin ^[2] ; Jiewei,Luo ^[2] ; Jianbin,Huang ^[3] ; Zhao,Jin ^[2]

作者单位： Department of Traditional Chinese Medicine, The First Affiliated Hospital, Fujian Medical University, Fuzhou, P. R. China. ^[1] Shengli Clinical Medical College of Fujian Medical University, Fuzhou, P. R. China. ^[2] Department of Traditional Chinese Medicine, Fujian Provincial Hospital, Fuzhou, P. R. China. ^[3]

关键词 AS-IV Bcl2 H9C2(2-1) cardiomyocytes autophagy microRNA-34a pAKT/AKT

主题词动物(Animals);自噬(Autophagy);细胞系(Cell Line);细胞存活(Cell Survival);剂量效应关系, 药物(Dose-Response Relationship, Drug);葡萄糖(Glucose);微RNAs(MicroRNAs);肌细胞, 心脏(Myocytes, Cardiac);氧化性应激(Oxidative Stress);大鼠(Rats);皂苷类(Saponins);三萜类(Triterpenes)

DOI 10.1080/21691401.2019.1687492

PMID 31713440

发布时间 2020-03-23

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Artificial cells, nanomedicine, and biotechnology

2019年47卷1期

4172-4181页

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Astragaloside-IV protects H9C2(2-1) cardiomyocytes from high glucose-induced injury via miR-34a-mediated autophagy pathway.

Artificial cells, nanomedicine, and biotechnology

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Artificial cells, nanomedicine, and biotechnology

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Astragaloside-IV protects H9C2(2-1) cardiomyocytes from high glucose-induced injury via miR-34a-mediated autophagy pathway.

Artificial cells, nanomedicine, and biotechnology

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Artificial cells, nanomedicine, and biotechnology

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